DHEA, a neurosteroid, decreases cocaine self-administration and reinstatement of cocaine-seeking behavior in rats

Ravid Doron, Lilach Fridman, Iris Gispan-Herman, Rachel Maayan, Abraham Weizman, Gal Yadid

פרסום מחקרי: פרסום בכתב עתמאמרביקורת עמיתים

תקציר

Dehydroepiandrosterone (DHEA), which can act as a potential antidepressant in both animals and humans, appears to lower distress involved with cocaine withdrawal. In fact, a role for neurosteroids in modulation of substance-seeking behavior is becoming increasingly clear. Therefore, we tested the effects of DHEA on the self-administration of cocaine (1 mg/kg/infusion) by rats. At maintenance, a relatively low dose of exogenous DHEA (2 mg/kg; i.p.) attenuated cocaine self-administration after several days of chronic treatment. More than 2 weeks (19 days) of daily DHEA injections were required to decrease the cocaine-seeking behavior of rats to less than 20% of their maintenance levels. DHEA does not seem to decrease cocaine self-administration by increasing the reinforcing properties of the drug, as indicated by a cocaine dose-response determination. After being subjected to extinction conditions in the presence of DHEA, rats demonstrated a minimal response to acute exposure to cocaine (10 mg/kg), which indicated a protective effect of DHEA on relapse to cocaine usage. Our results suggest a potential role for the neurosteroid DHEA in controlling cocaine-seeking behavior, by reducing both the desire for cocaine usage and the incidence of relapse.

שפה מקוריתאנגלית
עמודים (מ-עד)2231-2236
מספר עמודים6
כתב עתNeuropsychopharmacology
כרך31
מספר גיליון10
מזהי עצם דיגיטלי (DOIs)
סטטוס פרסוםפורסם - 7 אוק׳ 2006
פורסם באופן חיצוניכן

הערה ביבליוגרפית

Funding Information:
This study was supported in part by a grant from the Israel Science Foundation and from the Israel Anti-Drug Authority to GY. The research reported in this paper was completed as part of the first author’s PhD dissertation, in the Faculty of Life Sciences, Bar-Ilan University, Ramat-Gan, Israel.

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